Search the Community

But it seems to really be everything. Despite only being subscribed to the sleeve forums via my profile, I see lapband and bypass threads in my unread threads.

I just had my RNY gastric bypass on 9/17/15. I would really like a buddy to share journeys with. The more the merrier!!

Doctors in the South, Florida area are starting not to do the lap band procedures anymore. Their saying, its having too many erosions and revisions. Ugh!! Anyhow, I notice in other states within the US they no longer doing lapbands. Anyone else is knows of this too?

I'd seen a co-worker achieve amazing success with her bypass WLS. I asked her about it & she told me who she went to & what she had done. I then researched the various available WLS options; Bypass, RNY, Banding, DUO Switch (or whatever that's called); each procedure's benefits, fallbacks, possible complications, the actual procedure details, the risks, how many die on the table from each WLS procedure, and each year from post-opcomplications, the prognoses on each procedure, pre-op protocol, avg hospital stay, post-op protocol & long term prognoses. When I decided on the banding, I conducted a parallel analysis between the Lap & Realize bands (I chose the Realize). Then, I checked out the surgeons' practices, online. Read every page. I read every detail about each surgeons' Education, Colleges/Universities attended and whether they earned multiple degrees/ other specializations, their college ranking (if they're dead last in their class, I don't want em!) whether or not they graduated with honors or were awarded Fellowships (only the best of the best are), their affiliations & memberships & what boards/ committees they're serving on, whether or not they are graduate-school, teaching doctors, their practice histories (whether or not they've moved around a lot-not a good indication of quality), other specializations, their WLS philosophies & if they stay abreast of latest technologies, their success rate, if they participate with my ins. & what's covered / what's not, their surgery schedules (how far out they're booking surgeries), etc. Then, I went to Health Check site which shows patient-rating scores on a number of patient-driven experiences with their surgeon, plus with commentary. This site covers docs across a broad range of specializations, not just WLS. The site also shows whether the surgeon has been sued for malpractice or sanctioned by their licensing agency/oversight org. - for any number of possible violations. I found Malene Ingram to be an excellent fit for me & she was. After the surgery, though, you only see the PA's for fills, etc. I imagine the PA's refer more serious issues back to Doc Ingram, if it looks like more surgery is necessary.

Here is an academic overview of the various bariatric procedures with a bit of excess 'science stuff' thrown in. No opinion. No bias. Published 2012 by the UK Royal College of Physicians. If anyone requires further clarification to the sources contained (hopefully its been copied successfully), please see the reference list at the end. This will provide you (licensing permitting) with a link to those original source documents so you can do your own further research/analysis. Any questions or queries, please do not hesitate to ask. Revs x Overview of bariatric surgery for the physician Keng Ngee Hng, Specialty registrar in gastroenterology1⇓ and Yeng S Ang, Consultant gastroenterologist and honorary lecturer2 + Author Affiliations 1Salford Royal NHS Foundation Trust 2Faculty of Medicine, University of Manchester, Oxford Road, Manchester Address for correspondence: Dr KN Hng, 4 Fern Close, Shevington, Wigan WN6 8BL. Email:keng_ngee@hotmail.com Abstract The worldwide pandemic of obesity carries alarming health and socioeconomic implications. Bariatric surgery is currently the only effective treatment for severe obesity. It is safe, with mortality comparable to that of cholecystectomy, and effective in producing substantial and sustainable weight loss, along with high rates of resolution of associated comorbidities, including type 2 diabetes. For this reason, indications for bariatric surgery are being widened. In addition to volume restriction and malabsorption, bariatric surgery brings about neurohormonal changes that affect satiety and glucose homeostasis. Increased understanding of these mechanisms will help realise therapeutic benefits by pharmacological means. Bariatric surgery improves long-term mortality but can cause long-term nutritional deficiencies. The safety of pregnancy after bariatric surgery is still being elucidated. Introduction Obesity is a worldwide pandemic,1–4 with the number of obese children and adolescents increasing alarmingly.5 This has serious health and socioeconomic implications due to the attendant increase in related comorbidities.1,2,4,6 Obesity causes type 2 diabetes, hypertension, dyslipidaemia, cardiovascular disease, obstructive sleep apnoea, obesity hypoventilation syndrome, cancer, steatohepatitis, gastro-oesophageal reflux, gallstones, pseudotumour cerebri, osteoarthritis, infertility and urinary incontinence.1–6 Severe obesity reduces life expectancy by 5–20 years.1 Diet, exercise and drug treatments for severe obesity have been disappointing.1–3,5–12 At the present time, bariatric surgery is the only treatment that reliably produces substantial and sustainable weight loss.1–7,9,13 It is indicated in people with BMI >40 kg/m2 or with BMI >35 kg/m2 in the presence of significant comorbidity.3,5,7,14 Bariatric surgery is cost effective,3,6,15 achieving weight loss, as well as improvement or resolution of associated comorbidities.1,2,5,6,9,15,16 In the past decade, the development of centres of excellence,5,6laparoscopic techniques,2,5,6 improved safety profiles2,6,9 and better documentation of clinical effectiveness2,6,15 have fuelled an increase in the number of procedures performed. Types of surgery Bariatric surgical procedures are traditionally classified as restrictive, malabsorptive or combined according to their mechanism of action. The procedures most commonly performed are laparoscopic adjustable gastric banding and roux-en-y gastric bypass.3,13 Sleeve gastrectomy is increasingly performed.2,6,7 Biliopancreatic diversion and biliopancreatic diversion with duodenal switch are much more complex and performed infrequently.2,5,17,22 Other historical procedures are no longer in common use. In addition to restriction and malabsorption, recent evidence suggests that neurohormonal changes are an important effect of bariatric surgery.2,6,7,17,18 Bariatric surgery is only part of the management of severe obesity. Careful patient selection and preparation are extremely important, as are long-term compliance with diet, nutritional supplementation and follow up.2,5,6,19 Laparoscopic adjustable gastric banding2 A purely restrictive procedure, laparoscopic adjustable gastric banding (LABG) is the least invasive procedure, is completely reversible and has the lowest mortality.19 A silicone inflatable band is placed around the stomach cardia immediately below the gastrooesophageal junction (Fig 1). This is connected to a subcutaneous port that is used for band adjustment.5,6 The band compresses the cardia to generate a sense of satiety and reduced appetite, which is thought to be mediated via vagal afferents.2 Roux-en-Y gastric bypass (RYGB) is a combined procedure that is also performed laparoscopically. A 20–30-ml gastric pouch connected to the jejunum forms the Roux limb (Fig 2). The disconnected duodenal limb is anastomosed 75–150 cm along the Roux limb, forming a Y configuration. The distal stomach, duodenum and part of the proximal jejunum are thus bypassed.5–7,20 Despite the traditional classification of the this procedure, malabsorption is not significant with the standard RYGB surgery.7 In an extended gastric bypass, the Roux limb is lengthened to increase the malabsorptive component.6,20 In sleeve gastrectomy, 60–80% of the stomach is removed along the greater curvature to leave a restricting ‘sleeve’ of stomach along the lesser curve (Fig 3).5,20,21 Originally the first step of the biliopancreatic diversion with duodenal switch (see below), sleeve gastrectomy has evolved into a staging procedure for super obese or high-risk patients.2,5–7,20 It is also increasingly used as a standalone procedure.2,6,7 Biliopancreatic diversion (BPD) and biliopancreatic diversion with duodenal switch (BPD/DS) are malabsorptive operations that result in bypass of most of the small intestine. With BPD/DS, a sleeve gastrectomy is performed, leaving the pylorus intact. The duodenum is then disconnected and the stomach anastomosed to the distal small bowel (the ‘duodenal switch’), creating a short alimentary limb. The long biliopancreatic limb is then anastomosed to the ileum 75–100 cm proximal to the ileocaecal valve, so digestion and absorption occurs only in the short common channel.5,6,20 With BPD, a partial gastrectomy leaves a 400 ml gastric pouch and the common channel is shortened to just 50 cm.5 Safety profile and complications Bariatric surgery is safe.2,5,6,9,22–24 High-volume centres of excellence deliver bariatric surgery with inhospital mortality of 0.14% and 90-day mortality of 0.35%, which is comparable to that for cholecystectomy.6 Acute complications, including haemorrhage, obstruction, anastomotic leak, wound infection, cardiac arrhythmias, pulmonary emboli, respiratory failure and rhabdomyolysis, occur in 5–10% of patients.5,6,9,10,25 Long-term complications include internal hernias, anastomotic stenoses, marginal ulceration, fistulae, diarrhoea, dumping syndrome, gallstones, emotional disorders and nutritional deficiencies.5,6,13,15,20,25–27 Patients with LAGB can experience port problems, stomal obstruction, band slippage/erosion, pouch dilation, gastro-oesophageal reflux and oesophageal dilation.5,13,19,25 Malnutrition is a concern with BPD with or without DS.1,17 Long-term risks for sleeve gastrectomy are unknown.5 The Longitudinal Assessment of Bariatric Surgery22 reported overall 30-day mortality of 0.3% for 4,610 patients having LAGB or RYGB for the first time, with 4.3% of patients having a major adverse outcome within 30 days. This was most frequent among patients having open RYGB (7.8%). A meta-analysis involving 85,048 patients reported a total 30-day mortality of 0.28% and a two-year mortality of a further 0.35%.24 The most complex malabsorptive procedures had the highest perioperative mortality at 1.11%. Mortality for gastric banding is between one in 2,000 and one in 5,700.2 Effects on weight, comorbidities and long-term mortality After RYGB, patients lose 60–70% of their excess weight over two years, and this is largely durable.1,2,6,9,15,16,28 Weight loss is dependent on long-term compliance with dietary recommendations.2,5,6 Sugary, energy-dense foods and drinks can ‘bypass the bypass’. After gastric banding, patients lose about 50% (range 39%–59%) of their excess weight at a slower rate, often continuing into the fifth year.1,2,5,6,9,19 Regular band adjustment is necessary.16 The Swedish Obese Subjects (SOS) study reported a reoperation or conversion rate of 31% for gastric banding and 17% for gastric bypass among patients followed for ≥10 years, excluding operations for postoperative complications.16 However, at the centre in Melbourne, only about 10% of patients after LAGB need some revisional procedure, including band replacement, in the following 10 years.2 Biliopancreatic diversion with or without DS produces excess weight loss of 70.1%9sleeve gastrectomy produces initial excess weight loss of 55%, but this may not be durable.2 Bariatric surgery also produces significant improvement in obesity-related comorbidities, with the most remarkable effect being resolution of type 2 diabetes (T2DM). A meta-analysis encompassing 22,094 patients reported complete remission of T2DM in 76.8% of patients,9 and a registry from the UK with data on 8,710 patients reported resolution of T2DM in 85.5% of patients.29 Major improvement often occurs within days after RYGB, before significant weight loss is achieved.5–7,17,18 After LAGB, improvement in T2DM occurs more slowly as a result of weight loss.2,7,19 Combined or malabsorptive procedures produce greater improvement than purely restrictive procedures.1,2,5,9,18 Diabetes less than three years in duration, no insulin requirement, milder obesity with BMI <40 kg/m2 and weight loss ≥10% predict complete resolution of T2DM.18,19 Bariatric surgery is now advocated by some for the treatment of T2DM in patients with BMI <35 kg/m2.4,7,30,31 Bariatic surgery effectively treats all other associated comorbidities: from steatohepatitis and pseudotumour cerebri to urinary incontinence.1–3,5,6,15 Meta-analysis showed that hyperlipidaemia improved in ≥70% of patients, hypertension resolved in 61.7% (and resolved or improved in 78.5%) and obstructive sleep apnoea resolved in 83.6%.9 At five years, the risk of cardiovascular disease had decreased by 72%.3 The incidence of cancer also reduced markedly,6,15,16 as did the risk of developing new comorbid conditions.15 Long-term efficacy is well documented.5,6,28 At follow up after 10 years, the Swedish Obese Subjects (SOS) study showed a 29% reduction in adjusted all-cause mortality, primarily because of decreases in cancer and myocardial infarction.16A retrospective cohort study of 7,925 patients after RYGB reported a 40% reduction in all-cause mortality during mean follow up of 7.1 years.23 Specific mortality decreased by 56% for coronary artery disease, by 92% for diabetes and by 60% for cancer. A large observational study, in which the vast majority of patients had undergone RYGB, reported an 89% risk reduction in five-year mortality.15 Energy homeostasis and hormonal changes Weight loss after bariatric surgery is not explained by volume restriction and malabsorption alone.17 Indeed malabsorption is estimated to account for only 5% of the weight loss following standard RYGB.17 Bariatric surgery causes significant changes in the neurohormonal profile, which contributes to sustained weight loss through changes in appetite, satiety, food preferences and eating patterns and explains the remarkable effect on T2DM.2,5–7,17,18 The hypothalamus32 Hormonal signals provide information about energy status to the hypothalamus. Adipokines are secreted by adipose tissue and enterokines by the gut. Incretins are enterokines that stimulate release of insulin after food intake.18 Two hypothalamic circuits influence food intake, and both contribute to acquisition and storage of nutrient energy. The homeostatic circuit increases appetite and locomotion in response to energy shortage. The hedonic circuit is engaged at stable weight plateaus in association with increases in body fat. It heightens finickiness to taste of food. Obese animals overeat palatable food but undereat bland foods and lose weight. In our current obesogenic environment, the hedonic circuit facilitates the seeking of energy-dense foods uncoupled from energy status. Enterokines Ghrelin,33 which is mostly synthesised in the stomach, is a potent appetite stimulator involved in hunger and meal initiation. Circulating levels are inversely proportional to BMI and respond to changes in body weight. Ghrelin enhances gut motility and speeds gastric emptying.17 It promotes lipid accumulation and weight gain, favouring glucose utilisation. It also inhibits insulin secretion and impairs glucose tolerance.18 Levels of ghrelin reported after bariatric surgery have been variable, which may be due to differences in surgical techniques and research methods.7,18Overall, the trend is for a decrease in ghrelin levels after RYGB and an increase after gastric banding.7,17 Sleeve gastrectomy, which removes most of the ghrelin-producing stomach, reduces levels of ghrelin. Peptide YY (PYY)34 is secreted postprandially by L cells in the pancreas, small intestine and colon. It suppresses appetite and promotes satiety via signalling actions in the brain. It also delays gastric emptying (the ileal brake) and enhances insulin sensitivity.7 Secretion of PYY generally corresponds to the energy ingested, although it may vary depending on the macronutrient content.17Interestingly, levels also correlate positively with exercise intensity, with resulting decreases in food intake. Glucagon-like peptide 1 (GLP-1) is co-secreted postprandially with PYY in the distal intestine.17 A powerful incretin, GLP-1 potentiates glucose-stimulated insulin secretion, enhances β-cell growth and survival, inhibits glucagon release and enhances all steps of insulin biosynthesis.7,17 It also slows gastric emptying to produce greater gastric distension and helps regulate appetite and body weight.7,17 Obese individuals have lower levels of PYY and GLP-1, and levels are decreased further in patients with diabetes.5,17,18 Two hypotheses exist to explain the hormonal and metabolic effects of the RYGB: the hindgut hypothesis the foregut exclusion theory. The hindgut hypothesis postulates that after RYGB and malabsorptive procedures, rapid nutrient delivery to the distal gut L cells and their increased exposure to incompletely digested nutrients lead to an early and exaggerated PYY and GLP-1 response, contributing to early satiety, reduced meal size and early resolution of T2DM.7,17,18 Ileal transposition studies provide strong evidence for this. Interposition of an ileal segment into the proximal gut in rodents produced exaggerated PYY, GLP-1 and enteroglucagon responses, reduced food intake, weight loss, improved insulin sensitivity and overall improved glucose homeostasis.7 The foregut exclusion theory proposes that exclusion of the duodenum and proximal jejunum after RYGB is the mechanism that mediates the effects of bariatric surgery.7,18 However, duodenal–jejunal bypass experiments in rats supporting this theory are compounded by the accompanying pyloric disruption that results in accelerated gastric emptying and rapid nutrient delivery to the hindgut.7 The endoluminal duodenal–jejunal sleeve also accelerates gastric emptying by abolishing duodenal osmoreceptor control of pyloric contraction.7 This 60 cm-long sleeve prevents nutrient contact with the duodenum and proximal jejunum, while biliary and pancreatic secretions flow outside the sleeve, delaying digestion.35 A possible mediator of the foregut exclusion theory is the gastric inhibitory polypeptide or glucose-dependent insulinotropic polypeptide (GIP), which is secreted by K cells in the duodenum in response to nutrient absorption.18,39,40 In addition to its incretin action, GIP promotes lipogenesis,41 with GIP receptor knockout mice protected against diet-induced obesity and insulin resistance,39 while antagonism of the GIP receptor improves glucose tolerance and insulin sensitivity and partially corrects pancreatic islet hypertrophy and β-cell hyperplasia.40 Levels of GIP are suppressed after malabsorptive procedures.18,41 Adipokines Adiponectin17,36,37 is synthesised primarily in adipose tissue, with levels inversely correlated with BMI. It is an important insulin sensitiser, and hypoadiponectinaemia causes insulin resistance and T2DM. Adiponectin also possesses antiatherogenic, anti-inflammatory and cardioprotective properties and may act centrally to modulate food intake and energy expenditure. Weight loss following bariatic surgery increases levels of adiponectin. Leptin38,32 is secreted by adipose tissue and regulates body weight via its action on the hypothalamus. It increases nocturnally to stimulate lipolysis but also increases postprandially to induce anorexia. In addition, leptin plays an important role in glucose homeostasis. Levels of leptin are proportional to body fat, with starvation or energy shortage activating the homeostatic mechanism in the hypothalamus to restore energy balance. However, leptin resistance develops in obesity. Weight loss from bariatic surgery reduces leptin levels.17 Many other enterokines and adipokines exist, some of which may also play a part in producing and sustaining weight loss or diabetes remission after bariatric surgery.17,18 Understanding the mechanisms of action of bariatric surgery will help realise therapeutic benefits by pharmacological means.6,7,19 Nutritional deficiencies Nutritional deficiency is common after bariatric surgery and the risk increases from LAGB through SG and RYGB to BPD with or without DS.20,26,31 The problem is heightened by the fact that micronutrient deficiencies are already highly prevalent in obese patients before surgery.21 After surgery, patients are at particular risk of deficiencies in Vitamins B1, B12, C, A and D and folic acid, as well as Iron, Calcium and Protein.20,26 Lifelong prophylactic supplementation is often necessary, and regular monitoring is essential.26 Investigation of clinical syndromes resulting from malnutrition can be challenging. Anaemia20,27 After bariatric surgery, patients are prone to iron deficiency because of intestinal bypass, pouch hypoacidity and intolerance of red meat. Obesity creates a state of chronic inflammation that can contribute to anaemia. Anaemia can also be caused by deficiencies in folate, Vitamin B12, vitamin E (haemolytic anaemia), copper (anaemia and neutropenia), Vitamin A and zinc. In refractory anaemia, gastrointestinal blood loss must be considered. Bleeding in the excluded stomach, duodenum or biliopancreatic limb is problematic as the usual endoscopic access route is no longer available. Neurological problems6,20 Neurological symptoms can result from deficiencies in thiamine, vitamin B12, niacin, vitamin E and copper or from hypocalcaemia secondary to Vitamin D deficiency. Clinical syndromes includes Wernicke's encephalopathy, peripheral neuropathy, dry beriberi, neuropsychiatric beriberi, pellagra, ataxia, spasticity, myelopathy, muscle weakness, posterior column signs and ptosis. Oedema6,20 Patients with oedema may have underlying heart failure, which can also be due to wet beriberi (thiamine deficiency) or selenium deficiency. Hypoalbuminaemia may be caused by liver cirrhosis secondary to steatohepatitis; severe protein/calorie malnutrition; kwashiorkor; and diarrhoea secondary to bacterial overgrowth, malabsorption of bile salts and niacin deficiency. Eye, skin and hair problems20 Vitamin A deficiency causes difficulties with nocturnal vision and reduced visual acuity. Vitamin E deficiency can cause retinopathy. Thiamine deficiency can present with blurred or double vision. Dry skin, pruritus and rash can be caused by deficiencies in vitamin A, niacin, riboflavin, zinc and essential fatty acids. Hair changes can be due to zinc deficiency or protein malnutrition. Pregnancy after bariatric surgery About half of patients undergoing bariatric surgery are women of childbearing age,8 which introduces specific concerns. Obesity is strongly associated with infertility8 and increases the risk of obstetric complications.8 Yet the effects of rapid weight loss and potential malnutrition in pregnant patients are of concern. Bariatric surgery improves fertility42,43 and reduces the incidence of obesity-related complications such as gestational hypertension, gestational diabetes, pre-eclampsia and foetal macrosomia when compared with obese controls. The effect on premature delivery, miscarriage, intrauterine growth retardation, low birth weight and neural tube defects and the need for caesarean section are unclear.8,43 Maternal surgical weight loss reduces the prevalence of obesity and cardiometabolic risk factors in offspring until the adolescent years.42 Pregnancy seems to have little effect on the surgically induced weight loss.8 Patients are generally advised to delay pregnancy until after the period of maximal weight loss (12–18 months).8 Extra vigilance in preconception, antenatal and obstetric care is required. Conclusion In summary, bariatric surgery is a safe and effective treatment for severe obesity and its associated comorbidities. It is particularly effective in the treatment of T2DM. Neurohormonal changes that affect appetite, satiety, glucose homeostasis and long-term energy balance contribute to its long-term efficacy. Two hypotheses exist to explain how hormonal changes produce these effects, and both may contribute. Patient adherence to postsurgical aspects of management is very important. Pregnancy after bariatric surgery brings additional considerations. Finally, the indications for bariatric surgery are being widened. Acknowledgements Dr Keng Ngee Hng is a specialty registrar in gastroenterology and has previously submitted part of this work for her Master of Science in gastroenterology (Salford University). Dr Yeng S Ang is the educational supervisor for Dr Hng and has refined the ideas, concepts and layout of the previous work. Recent updates are also included within this paper. © 2012 Royal College of Physicians

Parisshel, You mentioned: I just read a lapband blog where the blogger decided her weight loss wasn't "fast enough" so she was going to get a fill that would allow her to take in her calories by liquid only. I just shook my head and thought "that is not the goal here." That sentence right there is the NUMBER ONE reasons for Lap Band complications. MANY here are losing their weight just like this, and THEY THINK they are in the "Clear" now because their bands has not turned on them -- at the moment. They think it is NORMAL to not be able to eat solids, If I KNEW that I would not suffer consequences by tightening my band up to allow ONLY liquids -- I would do this too, but I am not stupid, I've BEEN THERE AND DONE THAT APPROACH and I've seen too many others harmed by this approach, many NEWBIES that are less than 5 years post op will argue -- WELL nothing has happen to me - YET.....sometimes it takes awhile before horrible damage sets in. My advice, the lap band does not discriminate, it will bite and it will bite in a nasty way if the band is tighten beyond the recommended level. Dr. O'Brien put that safety chart out, yellow, green and red zones, describing HOW THE BAND WORKS clearly and MANY still IGNORE IT...and think they can get away with keeping a too tight band and think, it only happens to others....and sadly some don't care, they will just misuse the band until the reflux and vomiting get so bad until it has to be removed in an urgent way and then they will just revise to something else.... And if they revise to another surgery type.....Sooner or later these people will have to "comply" with SOME weight loss surgery, whether it be Band, Sleeve, Bypass or DS...sooner or later they will have to comply and these other surgeries will cost them their life if they don't comply...

@ itskariduh I'd like to befriend fellow gastric bypass patients share progress and see all your guys progress I should be getting mine this month or October at the latest my coordinator is really slowing things down for me :/

I had a revision from LAP Band to RNY. Only one dr in town would do it in one surgery. I wanted one surgery and my insurance would only pay for one surgery.

Ok, I myself am two weeks from being sleeved, so this is secondhand knowledge. One of the gals in my support group is 4 weeks out and she was just telling us how she drinks 1 beer at bowling and a glass of wine after work on occasion. My first instinct was, is she nuts? However, she is a well educated, successful woman who has had great success with her sleeve, so who am I to judge? My doctor and physiologist said they recommend waiting a year to reintroduce alcohol, simply because they want you to reach goal and too many times, we waste calories on drinks. Also, you metabolize alcohol differently after surgery. I am a revision so I do know this from having the lap band. I would be drinking a glass of wine, it went down fine, so I had another and BAM!!! I was hammered, and throwing up... Not good!! I will probably get backlash, and I am NO expert, but I think if you can handle it, use in moderation, and are getting your water and protein first, then have a drink:) cheers????

Here's a story! I was ON THE TABLE, put to sleep... SURGERY DAY (this was just in September) and they woke me up 20 minutes later and said "sorry, we can't operate. You have a huge mass in your stomach and we don't know what it is." YEAH. So I'm more-than-half drugged... confused... asking everyone who will look at me "DO I HAVE CANCER??" and getting answers like "we don't know what it is, yet. The doctor will talk with you when you are awake." I clearly remember replying OH I AM AWAKE NOW. They referred me to OBGYN and I found out a few days later I have a huge cyst on top of my uterus. I am heading back into surgery this coming Thursday for a hysterectomy and, if there are no surprises or complications, I will also receive my gastric bypass surgery at the same time (the two surgeons coordinated and are working together). So there's a story for you! : )

I believe the increased risk of kidney stones from bariatric surgery is in reference to gastric bypass. However obesity by itself is a cause for increased risk of the stones. Not drinking enough Water is considered one of the main causes. If you are diagnosed with a stone, it is advisable to be checked out by a Urologist as the size of the stone is extremely important as to the treatment required. You don't want to put your kidney in jeopardy.

Sometimes I wish I hadn't told so many ppl because some things annoy me like people only talking to me about it or saying things like "you've never been so thin before in your life" which isn't true.. I have been thinner I was thinner 2 years ago so it's not like impossible to remember so I kinda feel ppl feel forced to say something when I actually wished they didn't... My friends and family have been really supportive so I'm fine with ppl knowing i wanted to be honest with them but my mom and dad seem to have told everyone they know so I get random ppl I don't really know asking how I'm feel and how much I've lost and stuff when I don't even know their names... Also something that makes me wish ppl didn't know is the fact that in the past as soon as ppl started noticing weight loss and saying things something would change in my mind and I would soon break the diet or whatever and stop losing so ppl saying something just because they know makes me feel like that's gonna happen again... Anyway I wouldn't have done it without my family's support much less without them knowing, that would mean having to lie to them forever and I wouldn't be able to manage that...(no offense to ppl who decided not to tell theirs families but I'm my case I would never be able to pull that off)... My family has been really supportive and understanding and its nothin weird since my aunt had gastric bypass years ago and 2 of my cousins had their sleeves last year too also my dad is a doctor so he's pretty open to this kind of thing...

No perdon me la hise el bypass asen 7 semanas atras casi 8 empese a escribil en este sight asen UNOs Dias pero combo sea gracias todo as aqui son tan dulce gracias

Has anyone had to have the 24 hour PH test??? Did it help with approval for revision?

Well, what do you know, I am now a blogger. 24 hours from now I should have been banded and in recovery. My feelings at this time are all over the place. Anxious, brave, excited, scared. You name an emotion and I have felt it. If I'm not ashamed of using this tool, why are there so many people that are? I feel it is no different than any other tool, less bypass. I've been thru that surgery with my ex, a cousin, a sister, and a good friend, I only know 2 people that have been banded, 1 is an absolute star student, 1 who only lost 20 pounds she said as she had a hot dog in 1 hand and a slurpie in the other. I'm pretty much on my own, I think I must like it that way. I think my fear is change, with change usually comes chaos, and Lord knows I have had enough of that. I have decided to do this after losing 65 pounds on my own and gaining it back over 2 years. That's what pisses me off the most. That I thought I had the battle won, when in fact it had just begun.

Holla fellow bandsters! Hope you all had a fabulous Independence Day! I sat here and tried not to watch myself and boys have simultaneous combustion from the heat- alas we were all saved that scene. It was a strange holiday in that there was no BBQ to attend, here or otherwise, I didn't even go watch fireworks! I can't decide whether it was just too hot, or I'm just getting freaking too old to "OOOHH and AWWWW" about fire in the sky. Boys weren't interested either so I just listened to the neighbors pop it like it was hot til around midnight. My dogs didn't even seem to care. So the boys had Subway and I had the normal protein shake and cream of chicken soup but as an added bonus, I put strawberries and bananas with some Greek yogurt, 1 pkg Carnation sugar free instant breakfast and skim milk in the blender! Boys had smoothies I froze mine and had strawberry banana "ice cream" around 10 last night. Delish. So as you know, I have been thinking today I was scheduled to have my first fill. Thanks to this forum, I now know that the receptionist misspoke when she said "fill", what she meant to say was "Post-Op" visit. Regardless, I was excited to go pretty much anywhere at this point. I scrubbed up, brushed up and put on a comfy sundress to wear, good choice because I didn't have to worry about buttoning and or zipping anything, I'm still slightly swollen and all I had to do was put it over my head and Voila! I really had no clue what was going to happen at this 1st appointment and lucky me, I got a nurse who was clueless as well. The regular Nurse that takes care of such appointments was on vacation this week (HOW DARE SHE) so I don't know if she was brought up from another unit of the clinic/hospital or if she was an agency person, or if (Lord I hope not) she works there and I just never seen nor heard of her before. So kids this is how it went down. I was ushered into the examination room and sat on one of the chairs. The nurse says "Ok, so what have you been eating, and sorry, but I have to look at your incisions." Umm, Ok, I hope you're going to look at my incisions (DUH) and I told her protein shakes, yogurt, chicken soup, yada, yada, and then I said "Um, am I going to see the Dr today or what's happening here?" she said "Oh no honey, you won't see your Dr until after your 1 month check-up with the regular nurse who's on vacation, then 1 or 2 weeks after that appointment you will have 1 with the nutritionist, then the week after that you will have a fill" My head was spinning off my neck at that point. I decided it was best maybe not to ask not too many questions to this particular person at that particular time, besides, I have this forum if I want to know something right? Well you guessed it, I couldn't resist, I started asking because you know, I brought a small list. (Well I didn't want to forget anything ya know?) I looked at my little list and immediately mentally crossed off things that I assumed only the Dr would know, or I only personally wanted the Dr to answer. So I asked about vitamins I think, something along those lines and she excused herself and brought in a booklet and said "Did you get one of these?" Uh, I don't think so, it doesn't look familiar. So she handed it to me and then said she needed to look at my incisions, I said Ok, and she basically lifted my dress up and said those have to go and RRRIIIPPP off came the first one! HEY, I said. I don't know if I'm- RRRIIIPPP- well screw it guess I'm ready to release my surgi-strips. Look I know it sounds like a nightmare and it pretty much was, I could go on and on, but my point is I went to my post-op appointment and basically expected Ashton K. to jump out of somewhere telling me I've just been "Punked" although that didn't happen, I did get a a wink/half eye roll from the receptionist when the nurse was explaining to her the upcoming appointments I needed, especially when she called her Stephanie and she said "My name's Ashley" (insert half wink & eye roll) giggle. I guess I was pretty calm about the entire thing because when she took my blood pressure it was only 107/62. My temperature a chilly 97.3, and guess what? She never weighed me, and I didn't ask. I guess what I'm trying to say is that none of that really mattered. I already knew I wasn't going to be getting a fill today. I pretty much assumed that they were just going to check on me after surgery. I've survived the ripped off strips and now that I think of it, I'm glad she did it because I babied them so much and would have worn them as a badge as long as I could have and I need to move on. The receptionist, Ashley, and I agreed she would just call me on Monday and we would go from there, sounded great to me! On the way home I thought to myself, I'm not restricted at all. I could eat whatever I want right now and it would be fine with my twisty and my stomach. Maybe I'll just have a salad, that's not such a big deal. I thought like this for about 10 miles until I snapped out of it. Girl you better check yourself, for Christ's sake this is exactly why you had this done, if you don't start using it now, instead of thinking like a food addict, your road shall be even longer. I came home had a yogurt and a Crystal Light, I still wasn't even hungry, not really. I picked up the red booklet she gave me and it was "All you need to know about your upcoming gastric bypass surgery" I just laid it on my chest and laughed hysterically. As always, onward and upward and Lo & Behold....Velvet

I watched the eposide of Still Holding On and it was a Band over Bypass. I bet alot of people are saying a second gastric bypass. I have lots of people say I had a bypass....

Hi, I had my surgery 11/27/12 and I was 244 the day of surgery. I am now 213 and have been fluctuating between 216 & 213. I feel as tho I am doing something wrong. I log all I intent in myfitnesspal and barely make 700 cals. My NUT appt is next thus and I have only lost 4lb since the 27th and I am afraid. Is it normal? I am a .band to bypass revision.

Soy de Massachusetts y combo no yo me hise el bypass y estoy en mi 7 semana y esta fin de semana empieso stage5, pero soy nueva en Este sight UNOs Dias nada as escribes ingles se me ase mas fasil

I think we look at it as a tool to help us, where those that are not considering WLS surgery just believe that we will eat smaller amounts of whatever we want and everything will be fine. They have no concept of what actually happens. As soon as I am healed from this surgery, I will be having a very serious hip revision surgery. So serious, that my surgeon and his partner cancelled it the day of surgery saying that it needs to be done in a larger hospital with full access to all types of equipment. I had an appointment with the new ortho doc on 1/8 but as my RNY is now 1/4, I rescheduled for 3/1 to schedule the hip revision. I am hoping whatever weight I have lost by the time of that surgery will make it easier on me and the docs. Any surgery is complicated by obesity and I am doing this for my health, not my looks. I am 60-years-old and that simply does not matter anymore! People can say the darndest things, but we KNOW what we are really undertaking. Being thankful for RNYTalk and Obesityhelp for my only outside support! :wub: Blessings! Lynn

I am 65 years old. After gastric bypass surgery, my skin became extremely wrinkled, like paper crinkles. Overnight, I began to look a hundred years old. My wife suggested I use Bio-Oil available at CVS. I apply it once each morning and that takes care of the problem.

Dr. Wade Barker is one of a select few weight loss surgeons to perform both the LAP-BAND procedure and the Laparoscopic Gastric Bypass (Roux-en-Y) and he holds privileges at several Dallas/Ft. Worth hospitals including: Doctors Hospital, Pinecreek Medical Center, and Forest Park Medical Center. He has performed over 2500 bariatric surgery procedures at his center in Dallas/Ft. Worth with great success. http://adjingo.2cimple.com/web/camp/publishpreview/2962

Random drug tests? Dang, that's harsh. What are they gonna do if you test positive after the surgery? Go back in and reconnect your bypass??

I've struggled with depression, self image issues, and mental illness most of my life and I have crazy social anxiety. I don't believe that changing the outside will make me any more comfortable with myself. I hate shopping because being around people spikes my anxiety. I do most of my shopping at Wal-Mart because I can do it in the middle of the night when not many people are around and I can get everything I need in one place. To be completely honest, I don't care enough about myself to ever do this for me. My partner thinks it's an awful idea because she thinks surgery is drastic and I could lose weight on my own if I wanted to. My parents are kind of pushing it on me. Both of my parents had bypass surgery. My mom is skinny as all get out and my dad is now gaining weight and could easily be considered obese. I just want to live without pain so I can be with my partner Sent from my SM-J327P using BariatricPal mobile app

My "moment " came in May of 2018 with a series of events. I was 246 pounds, and didn't want to hit 250. I was wearing a 3x in clothes and they were getting tight, and I didn't know where I'd find clothes. I was out of breath and sweatty just walking to my car from work, and I had diabetes type 2. High blood pressure. I was literally a hot mess, and I said to myself "that's it!" I went to the informational session with my husband at my bariatric surgeon's office on May of 2018, completed Anthem's 6 months of requirements, and had my gastric bypass on March 27, 2019. Although I've had complications Im thrilled that I had the surgery! Sent from my SM-N960U using BariatricPal mobile app